齐云课题组
时间:2023-04-12 来源:

近五年发表文章:

1.     Salicylate Sodium SuppressesMonocyteChemoattractant Protein-1 Production by Directly InhibitingPhosphodiesterase3B in TNF-α-Stimulated Adipocytes. Int J Mol Sci. 2023, 24(1):320.

2.    Comparing the effects ofthree decoctions for coronavirus disease 2019 onsevere acute respiratorysyndrome coronavirus 2-related toll-likereceptors-mediated inflammations. JTradit Chin Med. 2023; 43(1):51-59.

3.     Resibufogenin, one ofbufadienolides in toad venom,suppresses LPS-induced inflammation via inhibitingNF-κB and AP-1 pathways. Int Immunopharmacol. 2022;113(PtA):109312.

4.     Oral administration ofForsythiasides-rich extractfrom Forsythiae fructus decreases the IgE productionthrough suppressing Th2response in shrimp protein-sensitized BALB/c mice.Phytomedicine Plus. 2022,2(4): 100358.

5.     A comparative study of spikeprotein of SARS-CoV-2and its variant Omicron (B.1.1.529) on some immunecharacteristics. Sci Rep. 2022, 12:17058.

6.     Anti-oxidative andanti-inflammatory activities ofthe ethanol extract of edible flower fromChimonanthus praecox. FrontPharmacol. 2022, 13:1004520.

7.    MAPKs/AP-1, not NF-κB, isresponsible for MCP-1 production inTNF-α-activated adipocytes. Adipocyte.2022,11(1):477-486.

8.    Aloe-emodin,a naturallyoccurring anthraquinone, is ahighly potent mast cell stabilizerthroughactivating mitochondrial calciumuniporter. Biochem Pharmacol. 2021, 186:114476.

9.    Forsythiasides-richextractfrom Forsythiae Fructusinhibits mast cell degranulation by enhancingmitochondrialCa2+ uptake.Front Pharmacol. 2021, 12:696729.

10.  SophoraflavanoneM, aprenylatedflavonoid from Sophora flavescens Ait., suppressespro-inflammatorymediatorsthrough both NF-κB and JNK/AP-1 signaling pathwaysin LPS-primedmacrophages. EurJ Pharmacol. 2021, 907:174246.

11.  肥大细胞在脂肪组织中的生理与病理作用. 中国病理生理杂志. 2021, 37(1): 151-158.

12.  代谢性炎症在代谢综合征中的作用. 生命科学. 2021, 33(1): 15-25.

13.  Amlexanoxexertsanti-inflammatory actions bytargeting phosphodiesterase 4Binlipopolysaccharide-activated macrophages. BBA- Molecular Cell Research.2020,1867:118766.

14. Isodeoxyelephantopin,asesquiterpene lactone from Elephantopus scaberLinn., inhibitspro-inflammatorymediators’ production through both NF-κB andAP-1 pathways in LPS-activatedmacrophages. Int Immunopharmal. 2020, 84:106528.

15.  Lucyoside B,a triterpenoidsaponinfrom Luffa cylindrica, inhibits the production ofinflammatory mediatorsvia bothnuclear factor-κB and activator protein-1pathways in activatedmacrophages. JFunct Foods. 2020, 69:103941.

16.  Qing-Kai-Ling injectioninducesimmediatehypersensitivity reaction via the activation of anaphylatoxinC3. FrontPharmacol. 2020, 10: 1524.

17.  Ethanol extract ofElephantopusscaber Linn.Attenuates inflammatory response via the inhibition ofNF-κBsignaling by dampening p65-DNA binding activity inlipopolysaccharide-activatedmacrophages.J Ethnopharmacol. 2020,25;250:112499.

18.  Penicillin causesnon-allergicanaphylaxis by activating the contact system. Sci Rep.2020,10(1):14160.

19.  肥大细胞在肠易激综合症中的作用研究进展.中国药理学通报. 2020, 36(2): 149-153.

20.  Canscoralucidissima, aChinesefolk medicine, exerts anti-inflammatory activities byinhibitingthephosphorylation of ERK1/2 in LPS-activated macrophages. BMC ComplementAlternMed. 2019, 19(1):371.

21.  经皮致敏小鼠肠道过敏模型的建立与评价. 中国实验动物学报. 2019, 27(5):619-625.

22.  Shuang-Huang-LianAttenuatesAirway Hyperresponsiveness and Inflammation in a ShrimpProtein-Induced MurineAsthma Model. Evid Based Complement Alternat Med. 2019:4827342.

23.  双黄连注射液对I型超敏反应性炎症的抑制作用. 中国药理学与毒理学杂志. 2018, 32(8): 627-633.

24.  注射用灯盏花素对脂多糖致巨噬细胞炎症损伤的抑制作用. 中国药理学与毒理学杂志. 2018, 32(10): 783-789.

25.  Shuang-Huang-Lianinjectioninduces an immediatehypersensitivity reaction via C5a but not IgE. SciRep.2018, 8(1):3572.

26.  Shuang-Huang-Lianpreventsbasophilic granulocyteactivation to suppress Th2 immunity. BMCComplementAltern Med. 2018,18(1):2.

27.  Dietary flavonetectochrysinexertsanti-inflammatory action by directly inhibiting MEK1/2 inLPS-primedmacrophages.Mol Nutr Food Res. 2018, 62(2), 1700288.

28.  Obacunone causessustainedexpression of MKP-1 thusinactivating p38 MAPK to suppresspro-inflammatorymediators throughintracellular MIF. J Cell Biochem.2018,119(1):837-849.


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